Publication | Open Access
A case of exercise-induced acute renal failure in a patient with enhanced renal hypouricaemia
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2000
Year
Glomerular DiseaseRenal PathologyPathologySerum Phosphate 4.5Renal FunctionKinesiologyExerciseHematologyApplied PhysiologyClinical ExerciseAcute Kidney InjuryChronic Kidney DiseaseHealth SciencesSodium HomeostasisKidney FailureClinical Exercise PhysiologyEnhanced Renal HypouricaemiaRenal PathophysiologyPotassium HomeostasisUrologyExercise PhysiologyPhysiologyBody TemperatureNephritic SyndromeAbnormalities.renal HypouricaemiaMedicineNephrologyKidney Research
112/60 mmHg and body temperature was 36.8°C.Physical examination did not reveal any abnormalities.Renal hypouricaemia is a rare condition caused by an Laboratory data were: haemoglobin 13.0 g/dl; haem-isolated defect in tubular uric acid transport, which is atocrit 38.9%; leukocyte count 7700/ml with normaltransmitted as an autosomal recessive trait [1]. The diVerentiation; platelet 231000/ml; total proteinmajority of cases have been reported in Japanese and 6.9 g /dl; serum sodium 143 mEq l; serum potas-non-Ashkenazic Jewish [2]. Two major complications sium 4.5 mEq /l; serum chloride 104 mEq l; serumin this disease are urolithiasis [3] and acute renal calcium 10.3 mg/dl; serum phosphate 4.5 mg/dl; BUNfailure (ARF) [4,5]. In 1989, Erley
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