Partial protection of N-acetylcysteine against MPA-capped CdTe quantum dot-induced neurotoxicity in rat primary cultured hippocampal neurons

Abstract

CdTe QD exposure caused death and apoptosis of rat primary cultured hippocampal neurons <italic>via</italic> generating reactive oxygen species and increasing intracellular calcium levels, which could be reversed by a common antioxidant NAC.