Corticosterone Transfer and Metabolism in the Dually Perfused Rat Placenta: Effect of 11β-hydroxysteroid Dehydrogenase Type 2

11β-Hydroxysteroid Dehydrogenase

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Glucocorticoid exposure in late gestation permanently programs rat hepatic phosphoenolpyruvate carboxykinase and glucocorticoid receptor expression and causes glucose intolerance in adult offspring.

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Placental 11β‐hydroxysteroid dehydrogenase: a key regulator of fetal glucocorticoid exposure

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Maternal Undernutrition during Late Gestation Induces Fetal Overexposure to Glucocorticoids and Intrauterine Growth Retardation, and Disturbs the Hypothalamo-Pituitary Adrenal Axis in the Newborn Rat¹

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Inhibition of 11β‐hydroxysteroid dehydrogenase, the foeto‐placental barrier to maternal glucocorticoids, permanently programs amygdala GR mRNA expression and anxiety‐like behaviour in the offspring

Leonie Welberg, Jonathan R. Seckl, Megan C. Holmes · European Journal of Neuroscience · 2000 · 347 citations