Effects of aspirin and Helicobacter pylori on the gastroduodenal mucosal permeability to sucrose.

Abstract

These results confirm the use of sucrose permeability as a marker of aspirin induced gastroduodenal mucosal injury and identify the stomach as the major site of increased permeability. H pylori infection does not seem to change gastric mucosal sucrose permeability either at baseline or after ingestion of aspirin.