Publication | Closed Access
Long-term amiodarone therapy: a cause of increased hepatic attenuation on CT
20
Citations
5
References
1984
Year
Molecular PharmacologyTherapeutic Drug MonitoringRadiologyHepatologyLong-term Amiodarone TherapyLiver PhysiologyPharmacologyHepatic AttenuationCt ScansClinical PharmacologyHepatotoxicityPharmacotherapyAnti-arrhythmic DrugLiver TransplantationMedicineDrug-induced Liver InjuryAnesthesiologyHealth Sciences
Approximately 40% of the anti-arrhythmic drug, amiodarone, consists of iodine. During long-term therapy both the drug and its major metabolite, desethylamiodarone, are deposited in the liver. CT scans of patients receiving such therapy demonstrated increased hepatic attenuation.
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