Influence of fluid resuscitation on renal microvascular PO2 in a normotensive rat model of endotoxemia

Abstract

The loss of kidney function during endotoxemia could not be explained by an oxygen deficiency. Renal oxygen redistribution could for the first time be demonstrated during fluid resuscitation. HES130/0.4 had no influence on the VO2,ren and restored renal function with the least increase in the amount of renal work.