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Restoration of Normal L-Type Ca <sup>2+</sup> Channel Function During Timothy Syndrome by Ablation of an Anchoring Protein

105

Citations

17

References

2011

Year

Abstract

We propose that AKAP150-dependent changes in Ca(V)1.2-LQT8 channel gating may constitute a novel general mechanism for Ca(V)1.2-driven arrhythmias.

References

YearCitations

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