Publication | Open Access

Involvement of AMPK, IKβα-NFκB and eNOS in the sildenafil anti-inflammatory mechanism in a demyelination model

InflammationNeuroinflammationSignal TransductionAutoimmune DiseaseMolecular PhysiologyMedicineSildenafil Anti-inflammatory MechanismImmunologyImmune RegulationAutoimmunityBrain-immune InteractionNeurologyImmunosuppressionDemyelination ModelNeuroimmunologySynaptic SignalingCell SignalingMolecular Signaling