Oxyntomodulin (glucagon-37 or bioactive enteroglucagon): A potent inhibitor of pentagastrin-stimulated acid secretion in rats

M Dubrasquet, D. Bataille, Christian Gespach

Bioscience Reports · 1982 · 102 citations · 10 references

Concepts

Abstract

Research Article| June 01 1982 Oxyntomodulin (glucagon-37 or bioactive enteroglucagon): A potent inhibitor of pentagastrin-stimulated acid secretion in rats M. Dubrasquet; M. Dubrasquet 1INSERM U10, Hôpital Bichat, 75877 Paris Cedex 18, France Search for other works by this author on: This Site PubMed Google Scholar D. Bataille; D. Bataille *Centre CNRS-INSERM de Pharmacologie-Endocrinologie, B. P. 5055, 34033 Montpellier, France Search for other works by this author on: This Site PubMed Google Scholar C. Gespach C. Gespach **INSERM U55, Hôpital St. Antoine, 75571 Paris Cedex 12, France Search for other works by this author on: This Site PubMed Google Scholar Author and article information Publisher: Portland Press Ltd Received: May 25 1982 Online ISSN: 1573-4935 Print ISSN: 0144-8463 © 1982 The Biochemical Society1982 Biosci Rep (1982) 2 (6): 391–395. https://doi.org/10.1007/BF01119301 Article history Received: May 25 1982 Views Icon Views Article contents Figures & tables Video Audio Supplementary Data Peer Review Share Icon Share Facebook Twitter LinkedIn Email Cite Icon Cite Get Permissions Citation M. Dubrasquet, D. Bataille, C. Gespach; Oxyntomodulin (glucagon-37 or bioactive enteroglucagon): A potent inhibitor of pentagastrin-stimulated acid secretion in rats. Biosci Rep 1 June 1982; 2 (6): 391–395. doi: https://doi.org/10.1007/BF01119301 Download citation file: Ris (Zotero) Reference Manager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu toolbar search search input Search input auto suggest filter your search All ContentAll JournalsBioscience Reports Search Advanced Search This content is only available as a PDF. © 1982 The Biochemical Society1982 Article PDF first page preview Close Modal You do not currently have access to this content.

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