Publication | Open Access

In vivo administration of D609 leads to protection of subsequently isolated gerbil brain mitochondria subjected to in vitro oxidative stress induced by amyloid beta-peptide and other oxidative stressors: Relevance to Alzheimer’s disease and other oxidative stress-related neurodegenerative disorders

Neurodegenerative DiseasesAlzheimer's DiseaseMitochondrial FunctionVitro Oxidative StressVivo AdministrationDegenerative PathologyNeuroprotectionNeurologyNeuroscienceNeurodegenerationAmyloid Beta-peptideMedicineCell BiologyOxidative StressProtective Mechanisms