Publication | Closed Access
Regulation and expression of <scp>IL</scp>‐32 in chronic rhinosinusitis
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Citations
29
References
2012
Year
The induction of IL-32 by TNF-α, IFN-γ and Th1 cells as well as its increased expression in sinus tissues from CRS patients with nasal polyps demonstrated a potential role for IL-32 in the pathogenesis of CRS.
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