Matrix Metalloproteinase-28 Deletion Exacerbates Cardiac Dysfunction and Rupture After Myocardial Infarction in Mice by Inhibiting M2 Macrophage Activation - Concepedia

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Matrix Metalloproteinase-28 Deletion Exacerbates Cardiac Dysfunction and Rupture After Myocardial Infarction in Mice by Inhibiting M2 Macrophage Activation

DOI Full Paper Access

207

Citations

44

References

2012

Year

Yonggang Ma, Ganesh V. Halade, Jianhua Zhang, Trevi A. Ramirez, Daniel Levin, Andrew Voorhees, Yu-Fang Jin, Hai‐Chao Han, Anne M. Manicone, Merry L. Lindsey

Circulation Research

Abstract

MMP-28 deletion aggravated MI-induced LV dysfunction and rupture as a result of defective inflammatory response and scar formation by suppressing M2 macrophage activation.

References

	Year	Citations

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