Ventricular tachycardia after in vivo DC shock ablation in dogs. Electrophysiologic and histologic correlation.

Abstract

Our results indicate that VT after DC shock ablation originates from cells with abnormal automaticity in the superficial subendocardial border zone around the central ablation lesion. Within 1 week edematous and necrotic cells in this border zone are replaced by a fibrotic layer, and this transition is associated with the disappearance of VT.