Publication | Closed Access
Absence of thrombospondin-2 increases cardiomyocyte damage and matrix disruption in doxorubicin-induced cardiomyopathy
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Citations
37
References
2011
Year
InflammationThrombosisCardiomyopathyHeart FailureMolecular PhysiologyMatrix DisruptionCardiovascular DiseaseMedicineDoxorubicin-induced CardiomyopathyThrombospondin-2 IncreasesPharmacotherapyCardiovascular ToxicityPharmacologyCell BiologyCardiology
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