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Inhibition of fatty acid amide hydrolase and cyclooxygenase-2 increases levels of endocannabinoid related molecules and produces analgesia via peroxisome proliferator-activated receptor-alpha in a model of inflammatory pain

InflammationPeroxisome Proliferator-activated Receptor-alphaCannabis UseAnti-inflammatoryHealth SciencesInflammatory PainAcute PainPain MedicineMedicinePhysiologyMolecular PainNeuropharmacologyPain ManagementPain MechanismCyclooxygenase-2 Increases LevelsPharmacologyPain Research