Publication | Closed Access

Exposure to bisphenol A during embryonic/fetal life and infancy increases oxidative injury and causes underdevelopment of the brain and testis in mice

Developmental BiologyBrain DevelopmentDevelopmental ToxicologyPhysiologyLipid PeroxidationEmbryonic/fetal LifeToxicologyNeuroprotectionFetal NeurodevelopmentExperimental ToxicologyMedicineToxicological MechanismOxidative StressHealth Sciences