Ulinastatin Improves Pulmonary Function in Severe Burn-Induced Acute Lung Injury by Attenuating Inflammatory Response

Abstract

These results indicate that the inhibitory effects of ulinastatin on inflammatory mediator production, neutrophil activation, and microvascular permeability are associated with the recovery of pulmonary functions in severe burn-induced acute lung injury and suggest that ulinastatin may serve as a potential therapeutic administration in critical burn care.