Publication | Closed Access
Icilin induces G1 arrest through activating JNK and p38 kinase in a TRPM8-independent manner
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Citations
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References
2011
Year
Molecular PharmacologyMolecular PhysiologySignal TransductionSignaling PathwayCell RegulationG Protein-coupled ReceptorP38 KinaseImmunologyIcilin Induces G1Trpm8-independent MannerCellular BiochemistryMedicineCell BiologyCell SignalingCellular Physiology
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