Publication | Open Access

A potent soluble epoxide hydrolase inhibitor, t-AUCB, acts through PPARγ to modulate the function of endothelial progenitor cells from patients with acute myocardial infarction

InflammationEndothelial Cell PathobiologyEndothelial Progenitor CellsCardiovascular DiseaseMedicineVascular PharmacologyMyocardial InfarctionImmunologyEndothelial DysfunctionVascular BiologyPharmacologyCell BiologyCardiologyAcute Myocardial InfarctionMolecular Signaling