Publication | Open Access
Mice devoid of prion protein have cognitive deficits that are rescued by reconstitution of PrP in neurons
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Citations
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References
2005
Year
Cognitive ScienceMolecular NeuroscienceMice DevoidPrion DiseaseCognitive DeficitsNeurologyNeurosciencePrion ProteinMolecular NeurobiologyMedicineSynaptic SignalingSocial Sciences
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