Publication | Closed Access

Intratracheal administration of mitochondrial DNA directly provokes lung inflammation through the TLR9–p38 MAPK pathway

InflammationIntratracheal AdministrationInflammatory Lung DiseaseMitochondrial FunctionLung InflammationImmunologyChronic InflammationMedicineCell BiologyCell SignalingMitochondrial DnaOxidative Stress