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Vascular ATP-sensitive potassium channels are over-expressed and partially regulated by nitric oxide in experimental septic shock

InflammationMolecular PhysiologyNeurophysiologyNitric OxideReactive Nitrogen SpeciePhysiologyEndothelial DysfunctionNitrosative StressIon ChannelsSepsisVascular BiologyElectrophysiologyMedicineExperimental Septic ShockSocial SciencesOxidative Stress