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Neoangiogenesis, T-lymphocyte infiltration, and heat shock protein-60 are biological hallmarks of an immunomediated inflammatory process in end-stage calcified aortic valve stenosis

Vascular DiseaseImmunologyPathologyImmunotherapyInflammationHeat Shock Protein-60AngiogenesisValve DiseaseCell TransplantationAutoimmune DiseaseAutoimmunityVascular BiologyAortic Valve StenosisCell BiologyTumor MicroenvironmentImmunomediated Inflammatory ProcessEndothelial DysfunctionValvular Heart DiseaseMedicine