<i><scp>S</scp>taphylococcus aureus</i>‐derived extracellular vesicles induce neutrophilic pulmonary inflammation via both <scp>T</scp>h1 and <scp>T</scp>h17 cell responses

Abstract

Staphylococcus aureus EV can induce Th1 and Th17 neutrophilic pulmonary inflammation, mainly in a TLR2-dependent manner. Additionally, S. aureus EV enhance the development of airway hypersensitivity to inhaled allergens.