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Amelioration of β-amyloid-induced cognitive dysfunction and hippocampal axon degeneration by curcumin is associated with suppression of CRMP-2 hyperphosphorylation

Neurodegenerative DiseasesMolecular NeuroscienceMedicineβ-Amyloid-induced Cognitive DysfunctionHippocampal Axon DegenerationCrmp-2 HyperphosphorylationNeuroprotectionNeuroscienceNeurologyNeurodegenerationDegenerative PathologyMolecular NeurobiologyPharmacologySynaptic SignalingSocial Sciences