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Notoginsenoside R1 attenuates amyloid-β-induced damage in neurons by inhibiting reactive oxygen species and modulating MAPK activation

Neurodegenerative DiseasesAlzheimer's DiseaseReactive Oxygen SpeciesMedicineNeuropharmacologyMapk ActivationDegenerative PathologyNeuroprotectionNeuroscienceNeurologyNotoginsenoside R1Molecular NeurobiologyPharmacologySynaptic SignalingOxidative StressProtective MechanismsNeuroinflammation