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Age-Dependent Renal Accumulation of 4-Hydroxy-2- Nonenal (HNE)-Modified Proteins Following Parenteral Administration of Ferric Nitrilotriacetate Commensurate with Its Differential Toxicity: Implications for the Involvement of HNE–Protein Adducts in Oxidative Stress and Carcinogenesis

Redox SignalingUrologyNitrosative StressIron MetabolismMedicineRenal InflammationPathologyAge-dependent Renal AccumulationToxicologyHne–protein AdductsRenal PathophysiologyMetabolismPharmacologyRedox BiologyNephrologyKidney ResearchToxicological MechanismOxidative Stress