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Long-Term caspase inhibition ameliorates apoptosis, reduces myocardial troponin-I cleavage, protects left ventricular function, and attenuates remodeling in rats with myocardial infarction

InflammationCardiac MuscleHeart FailureCardiovascular DiseaseLong-term Caspase InhibitionAtherosclerosisMedicineCell DeathCardiologyMyocardial Troponin-i CleavagePharmacologyCell BiologyCell SignalingAcute Myocardial InfarctionMyocardial Infarction