Publication | Closed Access

Hypothetical molecular mechanisms by which local iron overload facilitates the development of venous leg ulcers and multiple sclerosis lesions

Autoimmune DiseaseDisease MechanismMedicineImmunologyHematologyPathologyExperimental PathologyLocal Iron OverloadHypothetical Molecular MechanismsVascular BiologyMultiple Sclerosis LesionsMultiple SclerosisSclerodermaNeuroimmunologyCell BiologyConnective Tissue Disease