Changes in Capillary Shear Stress in Skeletal Muscles Exposed to Long‐Term Activity: Role of Nitric Oxide

Abstract

These data support a role for NO in the early elevation of capillary shear stress that initiates angiogenesis in stimulated muscles, likely via modulation of upstream vascular resistance, and could explain the lack of capillary growth in stimulated muscles when nitric oxide generation is suppressed.