Dopamine transporter binding in Gilles de la Tourette syndrome: a [¹²³I]FP‐CIT/SPECT study

Abstract

Patients with GTS show higher striatal binding of FP-CIT to the striatum in comparison with age- and gender-matched control subjects, indicating that dopamine transporter abnormalities are involved in the pathophysiology of GTS. These abnormalities appear to be distributed across both caudate and putamen.