Publication | Open Access
Chemically Inducible Inactivation of Protein Synthesis in Genetically Targeted Neurons
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2009
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Synaptic TransmissionNeurotransmitterAffective NeuroscienceShort-term PlasticityMolecular BiologyStructural PlasticityOptogeneticsSynaptic SignalingPsychologySocial SciencesNeural PlasticityInducible InactivationNeurodynamicsNeuromodulationDrug AddictionCognitive NeuroscienceNeurochemistryProtein DegradationCognitive ScienceMolecular NeuroscienceBehavioral NeuroscienceCortical RemodelingCell BiologySynaptic PlasticityNeurobiological MechanismNeurophysiologyNeurobiological FactorNeuroscienceSynaptic DysfunctionMolecular NeurobiologyCellular BiochemistryMedicine
Synaptic plasticity mediates a variety of physiological functions, including emotional regulation, drug addiction, and learning and memory ([Goelet et al., 1986][1]). There are two temporally distinct forms of synaptic plasticity: short-term plasticity, which lasts seconds to minutes, and long-term
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