Publication | Closed Access

An increase in spinal dehydroepiandrosterone sulfate (DHEAS) enhances NMDA-induced pain via phosphorylation of the NR1 subunit in mice: Involvement of the sigma-1 receptor

Pain SyndromeNr1 SubunitPain MedicineMedicineNeuropathic PainMolecular PainNeuropharmacologyNeurosciencePain MechanismPharmacologySigma-1 ReceptorNmda-induced PainPain Research