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Tetramerisation of α-latrotoxin by divalent cations is responsible for toxin-induced non-vesicular release and contributes to the Ca2+-dependent vesicular exocytosis from synaptosomes

Protein SecretionSynaptic TransmissionAnalytical UltracentrifugationSynaptic SignalingCellular PhysiologySecretory PathwayDivalent CationsBiochemistryIon ChannelsMembrane BiologyProtein TransportCell BiologyToxin-induced Non-vesicular ReleaseSignal TransductionNatural SciencesIntracellular TraffickingCellular BiochemistryMedicineCa2+-dependent Vesicular Exocytosis