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Enhanced vulnerability to oxidative stress by α-synuclein mutations and C-terminal truncation

Redox SignalingNeurodegenerative DiseasesBiochemistryNatural SciencesMolecular BiologyC-terminal TruncationDegenerative PathologyEnhanced VulnerabilityReactive Oxygen Specieα-Synuclein MutationsMedicineRedox BiologyMolecular MechanismsOxidative Stress