Publication | Closed Access

Dose–response transition from cell cycle arrest to apoptosis with selective degradation of Mdm2 and p21WAF1/CIP1 in response to the novel anticancer agent, aminoflavone (NSC 686288)

Nsc 686288Tumor BiologyMedicineApoptosisPharmacologyCancer Cell BiologyCell DeathCell Cycle ArrestAnti-cancer AgentCell BiologyCancer TreatmentTumor SuppressorOncologyProtein DegradationCell Death MechanismsDose–response TransitionCancer Research