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Both IgA nephropathy and alcoholic cirrhosis feature abnormally glycosylated IgA1 and soluble CD89–IgA and IgG–IgA complexes: common mechanisms for distinct diseases

Glomerular DiseaseGlomerulonephritisHepatologyLiver PhysiologyIga GlomerulonephritisRenal PathologyImmunologyPathologyAlcoholic Cirrhosis FeatureSoluble Cd89–igaHepatology InflammationAutoimmunityAutoantibodiesLiver DiseaseImmunologic DiseaseMedicineIgg–iga ComplexesCirrhosis