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Congenital Absence of Nitric Oxide Synthase 3 Potentiates Cardiac Dysfunction and Reduces Survival in Doxorubicin- and Trastuzumab-Mediated Cardiomyopathy

CardiomyopathyHeart FailureReduces SurvivalStructural Heart DiseaseCardiovascular DiseaseCongenital AbsenceMedicinePhysiologyTrastuzumab-mediated CardiomyopathyCongenital Heart DefectPharmacologyCardiologyCardiovascular GeneticsCardiac Pathology