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Modulation of Ca2+ channel currents in primary cultures of rat cortical neurones by amyloid β protein (1–40) is dependent on solubility status

Amyloid β ProteinSynaptic TransmissionNeurotransmissionCellular NeurobiologySynaptic SignalingCellular PhysiologySolubility StatusSocial SciencesHyperpolarization (Biology)NeurochemistryBiophysicsCa2+ Channel CurrentsIon ChannelsNeurodegenerative DiseasesNeurophysiologyPhysiologyNeuroscienceMolecular NeurobiologyMedicine