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Genetic variation in the two-pore domain potassium channel, TASK-1, may contribute to an atrial substrate for arrhythmogenesis

Cardiac MuscleChannelopathiesCardiomyopathyMolecular PhysiologySignal TransductionHyperpolarization (Biology)Atrial SubstratePhysiologyGenetic VariationElectrophysiologyCardiovascular PhysiologySystems BiologyMedicineCellular PhysiologyCardiac Mechanic