The <i>NOD2</i> defect in Blau syndrome does not result in excess interleukin‐1 activity - Concepedia

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The <i>NOD2</i> defect in Blau syndrome does not result in excess interleukin‐1 activity

DOI Full Paper Access

100

Citations

43

References

2009

Year

Tammy M. Martin, Zili Zhang, Paul A. Kurz, Carlos D. Rosé, Hong Chen, Huiying Lu, Stephen R. Planck, Michael P. Davey, James T. Rosenbaum

Arthritis & Rheumatism

Abstract

Taken together, these data suggest that in contrast to related IL-1beta-dependent autoinflammatory cryopyrinopathies, Blau syndrome is not mediated by excess IL-1beta or other IL-1 activity.

References

	Year	Citations

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