Publication | Closed Access

Cholesterol secoaldehyde induces apoptosis in H9c2 cardiomyoblasts through reactive oxygen species involving mitochondrial and death receptor pathways

Heart FailureOxysterolMitochondrial FunctionCardiovascular DiseaseBiochemistryAtherosclerosisPhysiologyApoptosisDeath Receptor PathwaysCell DeathH9c2 CardiomyoblastsMedicineLipid PeroxidationCardiovascular ToxicityReactive Oxygen SpeciesCardiologyOxidative Stress