Preservation of visual function in papilloedema observed for 3 to 6 years in cases of benign intracranial hypertension.

Abstract

PAPILLOEDEMA associated with a space-occupying lesion and raised intracranial pressure is known to lead to the development of optic atrophy within a few months unless the intra- cranial pressure is relieved with the minimum of delay (Huber, 1961). The atrophic changes are often accompanied by marked impairment of function as evinced by diminished visual acuity and by defects of visual field.