Publication | Open Access

Sustained augmentation of cardiac α1A-adrenergic drive results in pathological remodeling with contractile dysfunction, progressive fibrosis and reactivation of matricellular protein genes

Matricellular Protein GenesCardiomyopathyCardiac MuscleMolecular PhysiologyPhysiologyCardiac PathologyContractile DysfunctionCardiovascular PhysiologyProgressive FibrosisMedicineCell BiologyCardiologyCellular PhysiologyExtracellular MatrixMolecular Signaling