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A feedback loop in PPARγ–adenosine A2A receptor signaling inhibits inflammation and attenuates lung damages in a mouse model of LPS-induced acute lung injury

Acute Lung InjuryInflammationAnti-inflammatoryMolecular PhysiologyInflammatory Lung DiseaseLung InflammationMedicineImmunologyFeedback LoopPulmonary PharmacologyPulmonary FibrosisPparγ–adenosine A2a ReceptorPharmacologyCell SignalingMouse ModelPulmonary DiseasePulmonary Vascular Disease