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Attenuation of cardiac dysfunction by a PPAR-α agonist is associated with down-regulation of redox-regulated transcription factors

Molecular PharmacologyTranscriptional RegulationHeart FailureMolecular PhysiologyCardiomyopathyCardiovascular DiseasePpar-α AgonistMedicinePhysiologyCardiovascular PharmacologyCardiac DysfunctionCardiovascular PhysiologyCardiovascular FunctionPharmacologyCardiologyRedox-regulated Transcription FactorsOxidative Stress