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Cyclin-Dependent Kinase Inhibitors Attenuate Protein Hyperphosphorylation, Cytoskeletal Lesion Formation, and Motor Defects in Niemann-Pick Type C Mice

Cytoskeletal Lesion FormationSignal TransductionCell SignalingMedicineReceptor Tyrosine KinaseCell DeathCytoskeletonCell CycleMotor DefectsPharmacologyCell BiologyCellular PhysiologyProtein Phosphorylation