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Age-dependent high-density clustering of GM1 ganglioside at presynaptic neuritic terminals promotes amyloid β-protein fibrillogenesis

Synaptic PlasticityNeurodegenerative DiseasesAlzheimer's DiseaseMolecular NeuroscienceAmyloid β-Protein FibrillogenesisGm1 GangliosideProtein MisfoldingNeurologyNeuroscienceCell BiologyMolecular NeurobiologyNeuropathologyMedicineSynaptic SignalingAge-dependent High-density ClusteringSocial Sciences