Publication | Open Access

Gene targeting demonstrates that α4 nicotinic acetylcholine receptor subunits contribute to expression of diverse [3H]epibatidine binding sites and components of biphasic 86Rb+ efflux with high and low sensitivity to stimulation by acetylcholine

Molecular PharmacologyBiphasic 86Rb+BiochemistryLow SensitivityMedicineNatural SciencesG Protein-coupled ReceptorNicotineReceptor (Biochemistry)Molecular BiologyPharmacologySynaptic SignalingCell Signaling