Effects of Acute Ethanol Exposure on Regulatory Mechanisms of Bcl‐2‐Associated Apoptosis Promoter, Bad, in Neonatal Rat Cerebellum: Differential Effects During Vulnerable and Resistant Developmental Periods

Abstract

Ethanol-induced alterations of Bad-related mechanisms at P4 favor a prodeath response. EtOH does not influence these same mechanisms in a manner that promotes cell death at P7. Divergent Bad-related responses at these 2 developmental ages likely contribute to their differential EtOH vulnerability.